Mode of Action for Tribenuron-methyl Herbicide

Tribenuron-methyl is a selective sulfonylurea herbicide that targets broadleaf weeds in cereal crops like wheat, barley, and oats. Its mode of action involves inhibiting the enzyme acetolactate synthase (ALS), which is crucial for the synthesis of branched-chain amino acids (valine, leucine, and isoleucine) in plants.

1. Inhibition of ALS Enzyme: Tribenuron-methyl binds to the ALS enzyme, blocking its activity. This enzyme is essential for the biosynthesis of branched-chain amino acids, which are vital for protein synthesis and cell growth.
2. Cessation of Amino Acid Synthesis: The inhibition of ALS leads to a halt in the production of these essential amino acids, disrupting protein synthesis and cell division processes within the weed.
3. Plant Growth Stoppage: As a result, the affected weeds stop growing, showing symptoms such as yellowing and necrosis of leaves, eventually leading to plant death.
4. Systemic Action: Tribenuron-methyl is absorbed through the leaves and roots of the weed and is translocated throughout the plant, ensuring that all parts of the weed are affected.
5. Selective Toxicity: Due to its specific mode of action, tribenuron-methyl is highly selective, effectively controlling broadleaf weeds while being safe for cereal crops.

This herbicide provides long-lasting weed control with low application rates, making it an efficient and environmentally friendly choice for farmers.

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